Showing posts with label Syndrome. Show all posts
Showing posts with label Syndrome. Show all posts

Sunday, 3 July 2016

Injuries to the Median Nerve at the Wrist-Carpal Tunnel Syndrome-Injuries to the Median Nerve at the Elbow-Motor-Sensory-Vasomotor Changes-Trophic Changes-

Injuries to the Median Nerve at the Wrist
■■ Motor: The muscles of the thenar eminence are paralyzed and wasted so that the eminence becomes flattened. The thumb is laterally rotated and adducted. The hand looks flattened and “apelike.” Opposition movement of the thumb is impossible. The first two lumbricals are paralyzed, which can be recognized clinically when the patient is asked to make a fist slowly, and the index and middle fingers tend to lag behind the ring and little fingers.
■■ Sensory, vasomotor, and trophic changes: These changes are identical to those found in the elbow lesions.
Perhaps the most serious disability of all in median nerve injuries is the loss of the ability to oppose the thumb to the other fingers and the loss of sensation over the lateral fingers. The delicate pincer-like action of the hand is no longer possible
Carpal Tunnel Syndrome
The carpal tunnel, formed by the concave anterior surface of the carpal bones and closed by the flexor retinaculum, is tightly packed with the long flexor tendons of the fingers, with their surrounding synovial sheaths, and the median nerve. Clinically, the syndrome consists of a burning pain or “pins and needles” along the distribution of the median nerve to the lateral three and a half fingers and weakness of the thenar muscles. It is produced by compression of the median nerve within the tunnel. The exact cause of the compression is difficult to determine, but thickening of the synovial sheaths of the flexor tendons or arthritic changes in the carpal bones are thought to be responsible in many cases.
As you would expect, no paresthesia occurs over the thenar eminence because this area of skin is supplied by the palmar cutaneous branch of the median nerve, which passes superficially to the flexor retinaculum. The condition is dramatically relieved by decompressing the tunnel by making a longitudinal incision through the flexor retinaculum.

Injuries to the Median Nerve at the Elbow
Motor
The pronator muscles of the forearm and the long flexor muscles of the wrist and fingers, with the exception of the flexor carpi ulnaris and the medial half of the flexor digitorum profundus, will be paralyzed. As a result, the forearm is kept in the supine position; wrist flexion is weak and is accompanied by adduction.
The latter deviation is caused by the paralysis of the flexor carpi radialis and the strength of the flexor carpi ulnaris and the medial half of the flexor digitorum profundus. No flexion is possible at the interphalangeal joints of the index and middle fingers, although weak flexion of the metacarpophalangeal joints of these fingers is attempted by the interossei. When the patient tries to make a fist, the index and to a lesser extent the middle fingers tend to remain straight, whereas the ring and little fingers flex. The latter two fingers are, however, weakened by the loss of the flexor digitorum superficialis.
Flexion of the terminal phalanx of the thumb is lost because of paralysis of the flexor pollicis longus. The muscles of the thenar eminence are paralyzed and wasted so that the eminence is flattened. The thumb is laterally rotated and adducted. The hand looks flattened and “apelike.”

Sensory
Skin sensation is lost on the lateral half or less of the palm of the hand and the palmar aspect of the lateral three and a half fingers.
Sensory loss also occurs on the skin of the distal part of the dorsal surfaces of the lateral three and a half fingers. The area of total anesthesia is considerably less because of the overlap of adjacent nerves.

Vasomotor Changes
The skin areas involved in sensory loss are warmer and drier than normal because of the arteriolar dilatation and absence of sweating resulting from loss of sympathetic control.

Trophic Changes
In long-standing cases, changes are found in the hand and fingers. The skin is dry and scaly, the nails crack easily, and atrophy of the pulp of the fingers is present.

Tuesday, 28 June 2016

The Palm of the Hand-Skin-The Palmar Aponeurosis-Dupuytren’s Contracture-Carpal Tunnel Syndrome-

The Palm of the Hand
Skin
The skin of the palm of the hand is thick and hairless. It is bound down to the underlying deep fascia by numerous fibrous bands. The skin shows many flexure creases at the sites of skin movement, which are not necessarily placed at the site of joints. Sweat glands are present in large numbers.
The palmaris brevis is a small muscle that arises from the flexor retinaculum and palmar aponeurosis and is inserted into the skin of the palm. It is supplied by the superficial branch of the ulnar nerve. Its function is to corrugate the skin at the base of the hypothenar eminence and so improve the grip of the palm in holding a rounded object.
The sensory nerve supply to the skin of the palm is derived from the palmar cutaneous branch of the median nerve, which crosses in front of the flexor retinaculum and supplies the lateral part of the palm, and the palmar cutaneous branch of the ulnar nerve; the latter nerve also crosses in front of the flexor retinaculum and supplies the medial part of the palm.
The skin over the base of the thenar eminence is supplied by the lateral cutaneous nerve of the forearm or the superficial branch of the radial nerve.

The Palmar Aponeurosis
The palmar aponeurosis is triangular and occupies the central area of the palm. The apex of the palmar aponeurosis is attached to the distal border of the flexor retinaculum and receives the insertion of the palmaris longus tendon. The base of the aponeurosis divides at the bases of the fingers into four slips. Each slip divides into two bands, one passing superficially to the skin and the other passing deeply to the root of the finger; here each deep band divides into two, which diverge around the flexor tendons and finally fuse with the fibrous flexor sheath and the deep transverse ligaments.


The medial and lateral borders of the palmar aponeurosis are continuous with the thinner deep fascia covering the hypothenar and thenar muscles. From each of these borders, fibrous septa pass posteriorly into the palm and take part in the formation of the palmar fascial spaces . The function of the palmar aponeurosis is to give firm attachment to the overlying skin and so improve the grip and to protect the underlying tendons.

Dupuytren’s Contracture
Dupuytren’s contracture is a localized thickening and contracture of the palmar aponeurosis, which limits hand function and may eventually disable the hand. It commonly starts near the root of the ring finger and draws that finger into the palm, flexing it at the metacarpophalangeal joint. Later, the condition involves the little finger in the same manner. In long-standing cases, the pull on the fibrous sheaths of these fingers results in flexion of the proximal interphalangeal joints. The distal interphalangeal joints are not involved and are actually extended by the pressure of the fingers against the palm.

Surgical division of the fibrous bands followed by physiotherapy to the hand is the usual form of treatment. The alternative treatment of injection of the enzyme collagenase into the contracted bands of fibrous tissue has been shown to significantly reduce the contractures and improve mobility.

Carpal Tunnel Syndrome
The carpal tunnel, formed by the concave anterior surface of the carpal bones and closed by the flexor retinaculum, is tightly packed with the long flexor tendons of the fingers, with their surrounding synovial sheaths, and the median nerve. Clinically, the syndrome consists of a burning pain or “pins and needles” along the distribution of the median nerve to the lateral three and a half fingers and weakness of the thenar muscles. It is produced by compression of the median nerve within the tunnel. The exact cause of the compression is difficult to determine, but thickening of the synovial sheaths of the flexor tendons or arthritic changes in the carpal bones are thought to be responsible in many cases. As you would expect, no paresthesia occurs over the thenar eminence because this area of skin is supplied by the palmar cutaneous branch of the median nerve, which passes superficially to the flexor retinaculum. The condition is dramatically relieved by decompressing the tunnel by making a longitudinal incision through the flexor retinaculum.





























Injuries to the Bones of the Hand-Dislocation of the lunate bone-Fractures of the metacarpal bones-Bennett’s fracture-Compartment Syndrome of the Forearm--Volkmann’s Ischemic Contracture

Injuries to the Bones of the Hand
Fracture of the scaphoid bone is common in young adults; unless treated effectively, the fragments will not unite, and permanent weakness and pain of the wrist will result, with the subsequent development of osteoarthritis. The fracture line usually goes through the narrowest part of the bone, which, because of its location, is bathed in synovial fluid. The blood vessels to the scaphoid enter its proximal and distal ends, although the blood supply is occasionally confined to its distal end. If the latter occurs, a fracture deprives the proximal fragment of its arterial supply, and this fragment undergoes avascular necrosis.
Deep tenderness in the anatomic snuffbox after a fall on the outstretched hand in a young adult makes one suspicious of a fractured scaphoid.

Dislocation of the lunate bone
occasionally occurs in young adults who fall on the outstretched hand in a way that causes hyperextension of the wrist joint. Involvement of the median nerve is common.

Fractures of the metacarpal bones
 can occur as a result of direct violence, such as the clenched fist striking a hard object.
The fracture always angulates dorsally. The “boxer’s fracture” commonly produces an oblique fracture of the neck of the fifth and sometimes the fourth metacarpal bones. The distal fragment is commonly displaced proximally, thus shortening the finger posteriorly.

Bennett’s fracture
is a fracture of the base of the metacarpal of the thumb caused when violence is applied along the long axis of the thumb or the thumb is forcefully abducted. The fracture is oblique and enters the carpometacarpal joint of the thumb, causing joint instability.
Fractures of the phalanges are common and usually follow direct injury.


Compartment Syndrome of the Forearm
The forearm is enclosed in a sheath of deep fascia, which is attached to the periosteum of the posterior subcutaneous border of the ulna. This fascial sheath, together with the interosseous membrane and fibrous intermuscular septa, divides the forearm into several compartments, each having its own muscles, nerves, and blood supply. There is very little room within each compartment, and any edema can cause secondary vascular compression of the blood vessels; the veins are first affected, and later the arteries.
Soft tissue injury is a common cause, and early diagnosis is critical. Early signs include altered skin sensation (caused by ischemia of the sensory nerves passing through the compartment),
pain disproportionate to any injury (caused by pressure on nerves within the compartment), pain on passive stretching of muscles that pass through the compartment (caused by muscle ischemia), tenderness of the skin over the compartment (a late sign caused by edema), and absence of capillary refill in the nail beds (caused by pressure on the arteries within the compartment). Once the diagnosis is made, the deep fascia must be incised surgically to decompress the affected compartment. A delay of as little as4 hours can cause irreversible damage to the muscles.


Volkmann’s Ischemic Contracture
Volkmann’s ischemic contracture is a contracture of the muscles of the forearm that commonly follows fractures of the distal end of the humerus or fractures of the radius and ulna. In this syndrome, a localized segment of the brachial artery goes into spasm, reducing the arterial flow to the flexor and the extensor muscles so that they undergo ischemic necrosis. The flexor muscles are larger than the extensor muscles, and they are therefore the ones mainly affected. The muscles are replaced by fibrous tissue, which contracts, producing the deformity. The arterial spasm is usually caused by an overtight cast, but in some cases the fracture itself may be responsible. The deformity can be explained only by understanding the anatomy of the region.

Three types of deformity exist:
■■ The long flexor muscles of the carpus and fingers are more contracted than the extensor muscles, and the wrist joint is flexed; the fingers are extended. If the wrist joint is extended passively, the fingers become flexed.
■■ The long extensor muscles to the fingers, which are inserted into the extensor expansion that is attached to the proximal phalanx, are greatly contracted; the metacarpophalangeal joints and the wrist joint are extended, and the interphalangeal joints of the fingers are flexed.
■■ Both the flexor and extensor muscles of the forearm are contracted. The wrist joint is flexed, the metacarpophalangeal joints are extended, and the interphalangeal joints are flexed.

















Sunday, 19 June 2016

Cushing’s Syndrome-Addison’s Disease-Pheochromocytoma-Surgical Significance of the Renal Fascia-Aortic Aneurysms-Embolic Blockage of the Abdominal Aorta-

Cushing’s Syndrome
Suprarenal cortical hyperplasia is the most common cause of Cushing’s syndrome, the clinical manifestations of which include moon-shaped face, truncal obesity, abnormal hairiness (hirsutism), and hypertension; if the syndrome occurs later in life, it may result from an adenoma or carcinoma of the cortex.

Addison’s Disease
Adrenocortical insufficiency (Addison’s disease), which is characterized clinically by increased pigmentation, muscular weakness, weight loss, and hypotension, may be caused by tuberculous destruction or bilateral atrophy of both cortices.

Pheochromocytoma
Pheochromocytoma, a tumor of the medulla, produces a paroxysmal or sustained hypertension. The symptoms and signs result from the production of a large amount of catecholamines, which are then poured into the bloodstream. Because of their position on the posterior abdominal wall, few tumors of the suprarenal glands can be palpated. CT scans can be used to visualize the glandular enlargement; however, when interpreting CT scans, remember the close relationship of the suprarenal glands to the crura of the diaphragm.

Surgical Significance of the Renal Fascia
The suprarenal glands, together with the kidneys, are enclosed within the renal fascia; the suprarenal glands, however, lie in a separate compartment, which allows the two organs to be separated easily at operation

Aortic Aneurysms
Localized or diffuse dilatations of the abdominal part of the aorta (aneurysms) usually occur below the origin of the renal arteries. Most result from atherosclerosis, which causes weakening of the arterial wall, and occur most commonly in elderly men. Large aneurysms should be treated by open surgical repair. Endovascular repair can also be used by the introduction of a stent graft through one of the iliac arteries with access through the femoral arteries in the groin.

Embolic Blockage of the Abdominal Aorta
The bifurcation of the abdominal aorta where the lumen suddenly narrows may be a lodging site for an embolus discharged from the heart. Severe ischemia of the lower limbs results